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Later on, the molecular genetic basis of the disease was linked to a transition mutation in the snca gene A53T which results in the substitution of alanine to threonine in the N terminal 53rd amino acid of the protein leading to its abnormal aggregation (fibrillation) (Lee and Trojanowski, 2006)

Adipose tissue development and the molecular regulation of lipid metabolism

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This platform combines cyclic arginylglycylaspartic acid peptide (cRGD) with AuNPs via goldsulfur bonds to target HCC, NPs with cRGD-SH, and ultrasound-targeted microbubble destruction for reducing toxicity and achieving better targeted delivery of ATO and a nanoultrasound contrast agent (PFH)