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However, in the context of dysbiosis, this immune response is often exaggerated and prolonged, leading to chronic inflammation as reported [44]

On the other hand, mitochondria-related mechanisms of apoptosis activation prevail over the antiapoptotic mechanisms in this disorder [16, 158, 199]

To verify this result, we assessed the selective sensitivity of VRK2-KO cells (CFPAC-1 cells and SW1990 cells) to APR-017 or LCS3 by employing colony formation assays

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