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Cardiovascular disorders: PRLMs in inflammation resolution and tissue repair Following injury caused by ischemia, hypoxia, inflammatory mediators, physical trauma, chemical insults, biological infection, or drug-induced cardiotoxicity, the heart rapidly initiates an immune-inflammatory response to clear necrotic tissue and maintain homeostasis [4]

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Unlike many peptides that act through a single well-defined receptor, GHK-Cu exerts its biological effects through multiple mechanisms including direct stimulation of collagen and glycosaminoglycan synthesis, activation of antioxidant enzymes, modulation of metalloproteinase activity, promotion of angiogenesis, and broad regulation of gene expression across hundreds of genes involved in tissue repair, inflammation, and cellular protection