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Beyond SCFAs, arginine reinforces CD8 + T cell activity and inhibits Tregs via mTOR signaling (77), whereas Trp metabolites exhibit divergent effects: Kyn induces CD8 + T cell exhaustion through AhR-dependent programmed death-1 (PD-1) upregulation (78), contrasting with DATs enhancement of IFN-I-primed T cell expansion (79), and LPS exhibits dose-dependent immunomodulation, acutely activating T cells before promoting exhaustion during chronic exposure (80)

The observation that fusion of with Molt-3 cells was inhibited by pretreatment of intact with proteolytic enzymes [51] implies that this organism possesses a proteinase-sensitive receptor(s) responsible for binding and/or the establishment of tight contact with the cell surface of the host cell involved in fusion

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Reduces melanin production

[DOI] [PubMed] [Google Scholar] 23.Semenza GL