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For example, astrocyte-specific expression of SOCS3 in AD mouse models was shown to suppress not only GFAP induction and morphological hypertrophy but also neuroprotective and neurotoxic associated markers while simultaneously improving amyloid burden, synaptic function and cognition

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Zootec 51, e20220061

Its pro-apoptotic mechanism also involves suppression of fasciclin-like arabinogalactan protein-mediated Jun N-terminal kinase (JNK) signaling, leading to increased BAX/Bcl-2 ratio (31)

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