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Although current research indicates that ferroptosis plays a central regulatory role in the pathological process of FRDAlinking mitochondrial iron accumulation, neurodegeneration, and cardiomyopathyand provides a new theoretical anchor for understanding the FXN deficiencyiron dysregulationtarget-organ injury axis, several fundamental limitations remain

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The exception was observed in BALB/cByJ mice, where oxytocin administration reduced the distance traveled in the open field test [39]

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