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It should be noted that the examples herein were described using liposomal reduced glutathione that lack some of the preferred features of the invention

Furthermore, from a purely conceptual standpoint, one might postulate that metabolic reprogramming signals carried by these exosomes could disrupt the metabolic plasticity of recipient cells, blocking their adaptive transition from oxidative phosphorylation to glycolysis and thereby imprisoning cells in a metabolic phenotype highly sensitive to cuproptosis (99, 102)

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