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While TG serves as a physiological adaptation to excess energy intake, chronic overconsumption of SFAs disrupts lipid homeostasis, leading to ectopic TG accumulation in non-adipose tissues, a phenomenon known as lipotoxicity ( Exposure to SFAs drives macrophage polarization toward a pro-inflammatory M1-like phenotype

officinalis as the primary ingredient, sucrose as a sweetener, and other fruit juice concentrates as flavor enhancers

After ingestion, trazodone is metabolized in the liver by enzymes ( CYP3A4 ) into active and inactive compounds

Mitochondrial Dysfunction and Reactive Oxygen Species in Excitotoxicity and Apoptosis: Implications for the Pathogenesis of Neurodegenerative Diseases

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