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In the obese state, WAT undergoes selective rewiring of protein-protein interaction networks, resulting in interactions that are markedly enhanced or uniquely detectable in obese WAT [64]

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In glioma/glioblastoma (GBM), HDAC inhibitors targeting different subtypes exert distinct anti-tumor effects with diverse mechanisms: Class I HDAC inhibitors include Pyroxamide (HDAC1) and RGFP109 (HDAC1/3), both suppressing epithelial-mesenchymal transition (EMT) and invasion of GBM cells (Zhang et al., 2021