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Studies have shown that the stimulation of D4R on human T cells promotes quiescence (152), and overexpression of D4R in SLE patients may act as a compensatory mechanism to inhibit uncontrolled T cell proliferation, an important link in the pathogenesis of SLE

[DOI] [PubMed] [Google Scholar] 143.Sticha K.R., Sieg C.A., Bergstrom C.P., Hanna P.E., Wagner C.R