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It was withdrawn during phase 3 due to heart failure problems, but is still in clinical development for alternative indications

Overall, ATF1, ATF2, ATF4, ATF5 and ATF6 are persistently upregulated in most inflammatory, metabolic and stress-related diseases, acting primarily as transcriptional activators that drive pro-inflammatory programs or adaptive stress responses

In the next chapters, we will discuss how N-chlorinated proteins can confer protection and contribute to bacterial survival within the neutrophil phagosome, and on the other hand, how they modulate host immune responses to accelerate pathogen clearance