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Selenium inhibits oxidative stress-induced promotion of PCV2 replication by improving GPx1 expression, and overexpression of pig selenoprotein S inhibits OTA-induced promotion of PCV2 reproduction by inhibiting oxidative stress and p38 phosphorylation

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When this complex increases its content of nitrotyrosine and nitrosothiol groups in response to nitrosative stress, its activity is inhibited and therefore ATP production is diminished, which causes neuronal degeneration [10]

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