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Caspase-dependent HMGB1 release from macrophages participates in peripheral neuropathy caused by bortezomib, a proteasome-inhibiting chemotherapeutic agent, in mice

A previous study shows the overexpression of NOX1 in large and small bowel cancers in humans (105)

These interconnected mechanisms are summarized in Figure 1, which provides an overview of the effects of energy metabolism on the TMJ

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The same study also demonstrated that perturbing glutamine pathway either via glutamine depletion or by using specific chemical inhibitors or due to genetic polymorphism (e.g., GLS2, SLC7A5, and SLC1A5), decreased T-cell associated cytokine production by the macrophages (PMID: 30541099)