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They hypothesized that cysteine deprivation impairs the ETC activity and Krebs cycle, leading to hyperpolarization of the IMM followed by lipid peroxidation [230]

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Correspondingly, ACE2 or SLC6A19 deficiency in the small intestine decreases tryptophan absorption and intestinal antimicrobial peptide production, and increases susceptibility to IBD in mice [50]

When glutathione levels are supported, detox pathways tend to function more efficiently

The electrophilic nature of SFN drives its direct conjugation with glutathione, effectively depleting intracellular GSH pools and consequently inhibiting glutathione peroxidase 4 (GPX4) activity