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Similarly, the involvement of oxidative stress in sepsis, which disrupts redox signaling, leading to molecular damage, has been highlighted (172)

Candidate genes without the MADS domain were re-annotated manually with the assistance of FGENESH (Solovyev et al., 2006)

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Because of its high prevalence in conjunction with obesity, diabetes, and insulin resistance, NAFLD has been increasingly appreciated as a hepatic manifestation of metabolic syndrome and is the leading cause of death from liver disease in the United States 1,2,3

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