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Concurrently, non-oxidized dopamine lowered intracellular Fe 2+ accumulation by approximately 3040% at 25 M, decreased MDA production by about 2535% at 25 M, and sustained GSH levels restored by roughly 2030% compared with the erastin group

The overactivation of ACSL4, LPCAT3, and LOXs produces a large amount of phospholipid peroxides, which trigger ferroptosis

For BSO group, a week later, mice received intraperitoneal administration of 750 mg/kg of body weight BSO (B113387, Aladdin) in phosphate-buffered saline (pH 7.4) once a day for two weeks 25

Importantly, cell viability was not affected, meaning the compound did not simply kill fat cells but changed their metabolic behavior

So thats the other thing that I say