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Therefore, while ML162 treatment did not result in an enhancement of PSR-1 puncta localization at the tips of injured axons, it likely influenced the properties of PSR-1 condensates

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Experimental study demonstrates that LDN induces a phenotypic shift in BV-2 microglial cells, transitioning them from a pro-inflammatory(iNOS high CD206 low ) to a quiescent, anti-inflammatory M2 state (iNOS low CD206 high ) [150]

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