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In this case, GSH levels are well below the normal concentration, and therefore there is no feedback inhibition of GCL

CD38 mAb effects against MM can be maximized by combination therapies with immunomodulatory imide drugs (IMiDs), proteasome inhibitors (PIs), anti-programmed death 1 (PD-1)/programmed death ligand 1 (PD-L1) antibodies, or cellular therapies for the treatment of MM, especially in patients with relapsed or refractory MM (R/R MM) and drug-resistant MM

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Methylated forms are already in an active state, which may make them easier for the body to use compared to standard forms that require additional conversion