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Our results reveal that CoQ10 is potentially protective against METH-induced neurotoxicity in mice

Type I interferons promote the survival and proinflammatory properties of transitional B cells in systemic lupus erythematosus patients

Disruption of this process leads to the accumulation of dysfunctional mitochondria, increased oxidative stress, activation of inflammatory pathways, and progressive cellular dysfunction, thereby contributing to the pathophysiology of disorders such as endometriosis

doi: 10.1016/j.neurobiolaging.2012.05.009 48 HeY.WangJ.YingC.XuK

Another form of renal toxicity, generally associated with NSAIDs, is seen in patients with prerenal conditions leading to the reduction in renal blood flow or blood volume, where the renal prostaglandins have a supportive role in maintaining renal perfusion