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These mechanisms collectively maintain cell cycle homeostasis, thereby preventing cellular damage and aberrant proliferation

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The decrease of NCOA4 expression was significant in the late stage of AD pathological severity compared to control (Fig

The immune response to this damage, characterized by M1 macrophage polarization, neutrophilic inflammation, and Th17 activation, further exacerbates lipid peroxidation and ferroptosis, creating a vicious cycle that drives disease progression

Charoenwongpaiboon T, Punnatin P, Klaewkla M, Pramoj Na Ayutthaya P, Wangpaiboon K, Chunsrivirot S, Field RA, Pichyangkura R (2020) Conserved calcium-binding residues at the Ca-I site involved in fructooligosaccharide synthesis by Lactobacillus reuteri 121 inulosucrase