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A recent study conducted on mice models of PD established that the N-homocysteinylation of -SN enhances its aggregation potential and increases its neurotoxicity and that higher Hcy levels correlated with more severe -synuclein deposition within mouse brains (Zhou et al

Int J Biol Macromol 242:125101

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The COX-2 inhibitor parecoxib is neuroprotective but not antiepileptogenic in the pilocarpine model of temporal lobe epilepsy

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