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Replacing Cys with valine at these sites revealed that Cys304 mutation inhibited the transfer of Fas to lipid rafts, thereby blocking the function of death-related complexes and ultimately inhibiting apoptosis

Interestingly, the transcriptional expression of AtARR6 in OE lines was decreased under control conditions but was increased under cold stress compared with WT (Figure 8)

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endotoxin) can trigger systemic inflammation that further exacerbates neuroinflammation, illustrating how brain disorders are not confined to the CNS but involve a systemic network including the gut ecosystem [16]