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Heat Shock Protein 70 Inhibits Apoptosis in Cancer Cells through Simultaneous and Independent Mechanisms

Hansson E, Manjer J, Svensson H, Aberg M, Fagher B, Ekman R, Brorson H

Frontiers in Pharmacology, 14 , 1267765

NADPH contributes to recycling in oxidized antioxidant systems involving glutathione, thioredoxins, glutaredoxins, and peroxiredoxins and maintains another reactive oxygen species (ROS)-detoxifying enzyme, catalase, in its active form, suggesting that the homeostatic level of G6PD-derived NADPH determines the rate of oxidative damage [8]

These effects lead to more enhanced chemotherapy efficiency in a murine model of lung cancer 55 , underscoring the potential of NRF2 activation not only in widening the therapeutic window but also in actively reshaping the immune landscape within the TME