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Long-term observation studies have highlighted the ability of TLK199 to enhance bone-marrow maturation and cellularity

However, this does not explain the fact that, in the ER, the ratio of reduced to oxidized glutathione ([GSH]:[GSSG]) is optimal for disulphidebond formation (Lyles & Gilbert, 1991)

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This suggested that autophagy cargo receptors themselves, although increased at the protein level, are unlikely to be responsible for cell death following DC661 treatment