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The interaction of the severe acute respiratory syndrome coronavirus 2 spike protein with drug-inhibited angiotensin converting enzyme 2 studied by molecular dynamics simulation

Silencing PLCL1 with the NLRP3 inhibitor INF39 resulted in the downregulation of cytokine levels of IL-6, IL-1, and CXCL8, and therefore, INF39 could counteract the release of inflammatory cytokines caused by overexpression of PLCL1 (Luo et al., 2022

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In the next section, the mechanisms underlying this process will be discussed

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