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Off-label use in non-HIV populations relies on extrapolation, not large RCTs

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Yi C, Pan X, Yan H, Guo M, Pierpaoli W

58 Respiratory Syncytial Virus In in vitro modeling of RSV infection carried out in cultures of primary normal human bronchial epithelial cells, N-acetylcysteine significantly inhibited viral infection, re-established the number of beating cells, restored the virus-induced decrease in expression of -tubulin and genes involved in ciliagenesis such as DNAI2 and FOXJ1, inhibited IL-13, MUC5AC, and GOB5 upregulation, inhibited ICAM1 expression, increased heme-oxygenase 1 expression which correlated with the restoration of cellular antioxidant capacity, and enhanced intracellular hydrogen peroxide and glutathione concentrations

By supporting cellular survival cascades, it contributes to regenerative signaling environments in experimental models