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Regulation of ferroptosis-related signaling pathways in cancer drug resistance GPX4 and FSP1 signaling As mentioned earlier, GPX4 is a GSH-dependent ferroptosis inhibitor that reduces lipid peroxides, whereas FSP1 functions independently of GSH, which acts as an oxidoreduction enzyme reducing coenzyme Q (CoQ) to ubiquinol (CoQH2) at the cell membrane

95,96 8-oxoG can mispair with adenine and therefore leads to GT mutations during DNA replication

So thats just two generations, but its a remarkable and dramatic change in a short period of time

oxidativer und nitrosativer Stress, zum Beispiel durch Umwelteinflsse verschiedenster Art (Insektizide, Biozide, Abgase, Elektrosmog, Schwermetalle ) nicht optimale Ernhrung (Vitaminmangel, Alkoholkonsum ) Begleiteffekte (lebens-)notwendiger medizinischer Therapien (schdliche Abbauprodukte von Medikamenten, Nebenwirkungen von Strahlentherapien oder Chemotherapien ) Auerdem entstehen freie Radikale auch als Stoffwechselprodukt bei der normalen, gesunden Zellatmung