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For the delayed therapy, intraocular pressure was assessed as described above, at 48 h postL-NAME administration, before medication application, and thereafter at described intervals

These results suggest that increased tumor burden in the absence of host SPARC is a consequence of reduced collagen deposition, a disrupted vascular basement membrane, enhanced vascular function and an immune-tolerant, pro-metastatic microenvironment

132 MisraU

Although the physiological role of ferroptosis remains unclear, it can be triggered by pharmacological disruption of cellular lipid peroxidation repair or by oxidative stress associated with degeneration

[DOI] [PubMed] [Google Scholar] 12.Liscano Y, Oate-Garzn J, Delgado JP