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Pernicious anaemia is caused by a deficiency in vitamin B12, due to atrophic gastritis, an autoimmune condition (immune system destroys self), in this case the parietal cells of the stomach that produce intrinsic factor, a glycoprotein that binds to vitamin B12 in the stomach and is essential for normal absorption of vitamin B12 in the small intestine into the blood

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MFA mice treated with FAHF-2 did not experience anaphylaxis or change in body temperature following challenges with sensitized allergens [36]

Pre-treatment testing can determine which patients might benefit from combination anti-TNF-alpha and immunomodulator therapy[83,84]

Activation of human acid sphingomyelinase through modification or deletion of C-terminal cysteine