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It was found to inhibit MCA and TPA transformed cell growth via modulation of glutathione, mitogen-activated protein kinase and cancer pathways

PMID: 31801099

Abstract Pancreatic ductal adenocarcinoma (PDAC) presents significant treatment challenges, primarily due to its propensity for developing resistance to therapeutic interventions

Notably, the MDA-MB-231 line was a relative outlier compared to the tight correlation ( R 2 =0.997) for the other three lines (Supplemental Fig

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