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34, 35, 36 We extend these findings in a model of chronic tobacco exposure, which indicate the presence of systolic function impairment and that arbiters of this response include molecular pathways of increased inflammation and oxidative stress

M.HassonT.et al (1999)

The triple mutant clt1clt2clt3 is deficient in the transport of EC and glutathione from the plastid to the cytosol and shows lower cytosolic and higher plastidic glutathione levels 57 , but did not exhibit any difference in root length compared to WT when exposed to AITC

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