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Gomez DR, Cox JD, Roth JA, Allen PK, Wei X, Mehran RJ, Kim JY, Swisher SG , Rice DC, Komaki R

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however, as reducing NO accumulation via pharmacological inhibition of neuronal NO synthase, or genetic ablation of inducible NO synthase, failed to improve survival rates of mutant SOD1 transgenic mice

Protective effect of growth hormone-releasing hormone (GHRH) and its agonistic analog MR-409 in Alzheimers disease [abstract]

CNTNAP2 mutations may be associated with abnormal behaviors through alterations in synaptic neurotransmission, functional connectivity, and neuronal network activity