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188 On the one hand, as a strong vasoconstrictor produced by renal arteries, 20-HETE reduces the opening rate of calcium-activated potassium channels by activating G protein coupling and downstream PKC (protein kinase C) and MAPK pathways, 188 leading to enhanced vascular resistance of afferent arterioles, reduced glomerular capillary resistance and glomerular filtration rate, and resultant improvements in hypertension-induced renal end-organ damage