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Deficiency in Nrf2-GSH signaling impairs type II cell growth and enhances sensitivity to oxidants

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Its pharmacological effects primarily stem from its ability to suppress the expression and activity of inflammatory mediators triggered by liver injury, as well as reduce the production of ROS and nitric oxide (NO), thereby preserving antioxidants such as GSH (Tang et al., 2022)

Enteroendocrine cells (EECs) (such as enterochromaffin cells) can detect microbial signals and interact with afferent fibers of the vagus nerve to regulate gastrointestinal function by releasing hormones and neurotransmitters like serotonin (5-HT) and cholecystokinin (CCK) (, )

Lockwood SF, Gross GJ