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91 showed that PFD delayed OA progression by inhibiting the proliferation and migration of FLSs stimulated by TGF-1, while decreasing the expression Col1a1 , TIMP-1 , and ACTA-2 (-SMA), and IL-6 without affecting TNF- levels, thereby alleviating synovial inflammation and fibrosis, as evidenced by improved OARSI scores in OA models

Lets see how that affected its scoring

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Conversely, the conversion of Cu(II) to the more toxic Cu(I) induces cell death and intracellular oxidative stress, leading to an increase in heat shock protein 70 expression levels (190)

10.3389/fonc.2022.833814 41 LiL