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Identifying at-risk individuals and treating them in a prodromal phase based on their driving pathophysiological process could prevent the manifestation of PD, or at least alter the disease progression (Ascherio and Schwarzschild, At present, two approaches could be feasible to investigate this matter: the a-priori stratification of study cohorts (based on assumed mitochondrial dysfunction) or the in-vivo assessment of mitochondrial dysfunction (by neuroimaging methods or blood biomarkers) (Prasuhn et al., 2019)

Genetic defects in Indian Wilson disease patients and genotype-phenotype correlation

Secondly, development of specific iron uptake by release of phytosiderophores for chelating iron ( +++ when released into the rhizosphere

Pulmonary epithelial expression of human alpha1-antitrypsin in Transgenic mice results in delivery of alpha1-antitrypsin protein to the interstitium

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