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Palmitoylethanolamide (PEA), a lipid-derived peroxisome proliferator-activated receptor- (PPAR-), is shown to dismantle lipid droplets via -oxidation and restore innate cellular defenses 711

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In addition, the H 2 O 2 -induced activation of caspase-3 and the degradation of PARP were also blocked by glutathione administration concomitant with increased cell viability and reduced apoptosis