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Various metabolic pathways like fatty acid -oxidation pathway, oxidative stress, mitochondrial dysfunction, glycerophospholipid metabolism, tryptophan metabolism, and glutathione metabolism have been shown to play a role in neurodegenerative diseases (Widner et al

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GSH depletion by BSO treatment inhibits L2/vDNA trafficking from the Golgi to the mitotic chromosomes, resulting in accumulation of L2/vDNA within the Golgi of infected cells, low levels of nuclear L2/vDNA, and inefficient infection.

Due to the limited benefits of CLND and the significantly higher incidence of lymphedema following the procedure (258)