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The PI3K/AKT axis drives fibrotic responses via AKT/mTOR and PAK2/c-Abl signaling [43, 44], while the MEK/ERK pathway contributes to pulmonary fibrosis, particularly under Bmi-1 deficiency [45]

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However, GSSG supplementation did no longer lead to reduction of Grx1-roGFP2 in these strains, independent on whether the periplasmic binding protein GsiB or the permease domains GsiC or GsiD were missing, suggesting that Gsi is the exclusive transporter for oxidized glutathione in E