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These data suggest that suppression of NADPH oxidase activity by secreted effector proteins and toxins is not sufficient to defend these bacterial pathogens against oxidative attack and that these factors must work in concert with cellular processes critical for detoxification and repair to prevent restriction by host-derived ROS

Ecarnot (EA3920, University of Franche-Comt and University Hospital Besanon, France) for editorial assistance

Adv Exp Med Biol (2019) 1136:8795

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CIN low tumors exhibit a more pro-inflammatory environment, characterized by activated DCs and CD4 + T helper cells, whereas CIN high tumors contain anti-inflammatory macrophages, granulated MDSCs, and dysfunctional T cells [270]