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faecalis , FeCl 3 concentrations above 0.5 mM do not lead to further significant increases in intracellular iron levels (21)

4 Interplay between mitochondrial dysfunction and fatty acid metabolic disturbances in the failing heart Mitochondrial dysfunction and FAO disorders reinforce each other in the failing heart: ETC defects and m loss suppress FAO, while dysregulated FAO promotes oxidative stress, lipotoxicity, and structural damage, further compromising mitochondrial function

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Depressive symptoms could be relieved by N-methyl-d-aspartate (NMDA) receptor antagonists, group I metabotropic glutamate receptor (mGluR1 and mGluR5) antagonists, and positive modulators of -amino-3-hydroxy-5-methyl-4-isoxazole propionic acid (AMPA) receptors (78)

Modified neocortical and cerebellar protein expression and morphology following prenatal inhibition of the kynurenine pathway