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Additionally, indole-3-acrylic acid, through activation of the AhR, inhibits the TGF-/Smad pathway, thereby improving endothelial-mesenchymal transition in atherosclerotic mice (143)

doi: 10.1002/pld3.437 70

Interestingly, pre-treatment of cells with N-acetylcysteine, a precursor of glutathione, and a thiol antioxidant failed to block the depletion of glutathione, generation of ROS, and activation of caspase-3