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These results underscore, redox metabolism in senescence as a research area with actual therapeutic potential, and metabolomics as a powerful tool to move forwards in this field

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Western blots of Keap1, the negative regulator of Nrf2, show opposite effects, where Keap1 protein is highly expressed in both nave control limbs, and the low intensity control limb, as predicted with the low levels of Nrf2-ARE binding