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Glucose-6-phosphate dehydrogenase deficiency, the UDP-glucuronosyl transferase 1A1 gene, and neonatal hyperbilirubinemia

The metabolic shift from glucose utilization to ketone body consumption in malignant cells following KD implementation has been associated with enhanced AMPK stimulation [118,119,120,121,122]

Previous studies have only focused on the oxidative stress mechanism of CoNPs

Analysis of neurodevelopmental delay in children exposed in utero to hyperemesis gravidarum reveals increased reporting of autism spectrum disorder

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