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However, recent studies in metabolically competent HepaRG cell lines [23] and primary human hepatocytes [66] have confirmed that exposure to high levels of APAP result in GSH depletion, APAP protein adduct formation in mitochondria and organelle dysfunction accompanied by oxidant stress, resulting in necrotic cell death in human cells as demonstrated earlier in mouse models

Pharmacological inhibition of cystine-glutamate exchange induces endoplasmic reticulum stress and ferroptosis

Alanine Transaminase (ALT) and Alkaline Phosphatase (ALP): Elevated blood levels of these enzymes can be a sign of biliary obstruction