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Myeloid deletion of nuclear factor erythroid 2-related factor 2 increases atherosclerosis and liver injury

According to the A-tau interaction hypothesis, A and tau pathology, which results in synaptic dysfunction, neuronal death, and cognitive impairment (96), is determined by the intrinsic interaction with A and tau (A42 enhances tau phosphorylation, truncation, and aggregation, while tau can increase A synthesis)

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These differences may affect RBDs interaction with other molecules, including binding partners, as well as its overall biological activity