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While timely reperfusion remains the cornerstone therapeutic intervention, paradoxical reperfusion injury frequently manifests as blood-brain barrier (BBB) disruption, cerebral edema, and exacerbated neuronal damage (Guo et al., 2016), critically impacting patient prognosis

Notably, recent evidence highlights that aneuploid cancer cells depend on a mitotic G6PD-driven NADPH surge to neutralize ROS and ensure faithful chromosome segregation, and blocking this precipitates chromosomal instability [79]

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(72), CB-GLP-1 was designed based on intracerebral mitochondrial changes in patients with PD with the aim of ameliorating motor dysfunction

They are frequently utilized in drug delivery, with carbohydrates and proteins as subdivisions