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231 Given that altered regulation of Cu, homeostasis has been associated with a variety of disease conditions, future studies are warranted in order to determine the precise mechanism by which Cu imbalance causes cellular damage, thus providing important new information that can be used to design treatments for preventing disease and/or slowing disease progression

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Likewise, FGF21 administration inhibits formation of bleomycin-induced pulmonary fibrosis in vivo by suppressing oxidative stress via NRF2 activation, and inhibiting TGF--induced activation of pulmonary myofibroblasts (224)

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