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At the cellular level, excessive iodine directly inhibits thyroid peroxidase (TPO) activity and sodium-iodide symporter (NIS) expression, thereby disrupting thyroid hormone synthesis ( in vitro and in vivo studies delineates a coherent pathway from initial cellular damage and thyroid dysfunction to the breakdown of immune tolerance

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10.1074/jbc.R300008200 J

Third, emerging evidence suggests RVX possesses pleiotropic effects beyond anti-coagulation that may directly address the multiple pathways involved in IR injury pathogenesis 30,31