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However, equilibrium dialysis data 8 and direct EPR measurements 9 showed that HSA competes effectively with His for binding of Cu(II) at equimolar concentrations suggesting that excessive concentration of HSA in the blood serum should avoid the binding of Cu(II) to His and other free amino acids in the blood

Sikiric et al., 2020b), this provides overcoming (Seiwerth et al., 2018) of the technical and practical limitations now presented as shortage in the corresponding standard angiogenic growth factors applicability and diverse delivery and carrier systems (see Practical Application as Support )